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A list of all pages that have property "Description" with value "'''''N,N,N',N'''-Tetramethyl-''p''-phenylenediamine dihydrochloride, TMP". Since there have been only a few results, also nearby values are displayed.

Showing below up to 18 results starting with #1.

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     ('''''N,N,N',N'''-Tetramethyl-''p''-phenylenediamine dihydrochloride, TMP)
    • P50  + ('''''p''<sub>50</sub>''' is th'''''p''<sub>50</sub>''' is the oxygen partial pressure at which (a) respiratory flux is 50% of maximum oxygen flux, [[Jmax|''J''<sub>max</sub>]], at saturating oxygen levels. The oxygen affinity is indirectly proportional to the ''p''<sub>50</sub>. The ''p''<sub>50</sub> depends on metabolic state and rate. (b) ''p''<sub>50</sub> is the oxygen partial pressure at which oxygen binding (on myoglobin, haemoglobin) is 50%, or desaturation is 50%.n partial pressure at which oxygen binding (on myoglobin, haemoglobin) is 50%, or desaturation is 50%.)
    • Dinitrophenole  + ('''2,4-dinitrophenole''' (C<sub>6</sub>H<sub>4</sub>N<sub>2</sub>O<sub>5</sub>; M = 184.11 g·mol<sup>-1</sup>) is a protonophore acting as an [[uncoupler]] of [[oxidative phosphorylation]].)
    • 2-Deoxyglucose  + ('''2-Deoxyglucose''', also known as 2-deoxy-D-glucose is a glucose derivative that has the 2-hydroxyl group replaced by hydrogen. It competitively inhibits glycolysis by blocking hexokinase and phosphohexoseisomerase.)
    • 2-mercaptoacetate  + ('''2-mercaptoacetate''' is an inhibitor of'''2-mercaptoacetate''' is an inhibitor of medium-chain acyl-CoA dehydrogenase, MCAD, the rate-limiting enzyme of [[octanoylcarnitine]] oxidation. 2-mercaptoacetate has been used as an inhibitor of [[fatty acid oxidation]] ([[F-pathway control state]]). In permeabilized rat soleus muscle fibers, pre-incubation with 1 mM 2-mercaptoacetate for 45 min resulted in 58% inhibition of MCAD and decreased [[octanoylcarnitine]]&[[malate]] stimulated respiration by approximately 60% ([[Osiki 2016 FASEB J]]).[[Osiki 2016 FASEB J]]).)
    • O2k-Catalogue: O2k-Modules  + ('''4'''#### '''''O2k-Catalogue: O2k-Modules'''''. O2k-Modules can be obtained with the O2k or added later, and can be simply installed by the user.)
    • Tetrachloro-2-trifluoromethylbenzimidazole  + ('''4,5,6,7-Tetrachloro-2-trifluoromethylbenzimidazole''' is a protonophore or [[uncoupler]] of oxidative phosphorylation.)
    • HPTS  + ('''8-Hydroxypyrene-1,3,6-trisulfonic acid trisodium salt (HPTS)''' is a ratiometric pH fluorophore; pKa = 7.3. Relative molecular mass: ''M''<sub>r</sub> = 524.39)
    • AMPK  + ('''AMP-activated protein kinase''' is a regulatory protein which acts as crucial cellular energy sensor by sensing AMP, [[ADP]] and/or Ca<sup>2+</sup> levels in response to metabolic stresses or drug administration.)
    • ATP synthase  + ('''ATP synthase''' or F-ATPase (F<sub&g'''ATP synthase''' or F-ATPase (F<sub>1</sub>F<sub>O</sub>-ATPase; the use of Complex V is discouraged) catalyzes the [[endergonic]] phosphorylation of [[ADP]] to [[ATP]] in an over-all [[exergonic]] process that is driven by proton translocation along the [[protonmotive force]]. The ATP synthase can be inhibited by [[oligomycin]].[[oligomycin]].)
    • ATPases  + ('''ATPases''' are enzymes that hydrolyse [[ATP]]'''ATPases''' are enzymes that hydrolyse [[ATP]], releasing [[ADP]] and [[inorganic phosphate]]. The contamination of isolated mitochondria with ATPases from other organelles and endogenous adenylates can lead to the production of ADP, which can stimulate respiration. This situation would lead to an overestimation of [[LEAK respiration]] measured in the absence of ADP, ''L''(n) and subsequent inhibition of respiration by oligomycin, ''L''(Omy). of respiration by oligomycin, ''L''(Omy).)
    • About DatLab  + ('''About DatLab''' contains general information about DatLab 8, including the current version number and used licenses.)
    • Acceleration  + ('''Acceleration''', '''''a''''', is the ch'''Acceleration''', '''''a''''', is the change of [[velocity]] over time [m·s<sup>-2</sup>].</br> '''''a''''' = d'''''v'''''/d''t''</br>The symbol ''g'' is used for acceleration of free fall. The standard acceleration of free fall is defined as ''g''<sub>n</sub> = 9.80665 [m·s<sup>-2</sup>].ned as ''g''<sub>n</sub> = 9.80665 [m·s<sup>-2</sup>].)
    • Acclimation  + ('''Acclimation''' is an immediate time scale adaption expressing phenotypic plasticity in response to changes of a single variable under controlled laboratory conditions.)
    • Acclimatization  + ('''Acclimatization''' is an immediate time scale adaption expressing phenotypic plasticity in response to changes of habitat conditions and life style where several variables may change simultaneously.)
    • Acyl-CoA dehydrogenase  + ('''Acyl-CoA dehydrogenases''' ACADs are lo'''Acyl-CoA dehydrogenases''' ACADs are localized in the mitochondrial matrix. Several ACADs are distinguished: short-chain (SCAD), medium-chain (MCAD), and long-chain (LCAD). ACAD9 is expressed in human brain. ACADs catalyze the reaction</br>:::: acyl-CoA + FAD → ''trans''-2-enoyl-CoA + FADH<sub>2</sub>→ ''trans''-2-enoyl-CoA + FADH<sub>2</sub>)
    • Acyl-CoA oxidase  + ('''Acyl-CoA oxidase''' is considered as a '''Acyl-CoA oxidase''' is considered as a rate-limiting step in peroxysomal ''β''-oxidation, which carries out few ''β''-oxidation cycles, thus shortening very-long-chain fatty acids (>C<sub>20</sub>). Electrons are directly transferred from FADH<sub>2</sub> to O<sub>2</sub> with the formation of H<sub>2</sub>O<sub>2</sub>.t; to O<sub>2</sub> with the formation of H<sub>2</sub>O<sub>2</sub>.)
    • Acylcarnitine  + ('''Acylcarnitines''' are esters derivative'''Acylcarnitines''' are esters derivative of [[carnitine]] and [[fatty acid]]s, involved in the metabolism of fatty acids. Long-chain acylcarnitines such as [[palmitoylcarnitine]] must be transported in this form, conjugated to carnitine, into the mitochondria to deliver fatty acids for fatty acid oxidation and energy production. Medium-chain acylcarnitines such as [[octanoylcarnitine]] are also frequently used for high-resolution respirometry.tly used for high-resolution respirometry.)
    • Adaptation  + ('''Adaptation''' is an evolutionary time scale expression of phenotypic plasticity in response to selective pressures prevailing under various habitat conditions.)